通过分析56例SAH住院病人的心电图改变包括心律失常:窦速、窦缓、室早、室上速、室速及T波改变,S—T改变,Q—T延长,u波等。通过复习文献发现其发生机制为,SAH时其下丘脑受刺激使交感活动异常,使心脏的交惑神经局部释放的去甲肾上腺素增多所致,从而引起不同程度的心肌损害。推荐给SAH病人用β受体阻滞剂能帮助保存心肌功能,但有时可增加血管痉挛,而钙通道阻滞剂能减少血管痉挛。 The electrocardiogram changes in 56 inpatients with SAH included arrhythmia: sinus rate, sinus latency, ventricular rate, supraventricular tachycardia, ventricular tachycardia and T wave changes, S-T changes, Q-T prolongation, u wave and so on. By reviewing the literature found that the mechanism occurs when the hypothalamus is stimulated by SAH sympathetic abnormalities, the heart of the local nerves caused by the release of norepinephrine increased, causing varying degrees of myocardial damage. Beta-blockers recommended for SAH patients help preserve myocardial function, but sometimes increase vasospasm, whereas calcium channel blockers reduce vasospasm.