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宫内发育迟缓(IUGR)的发生与母体,胎盘胎儿的机能有着复杂的关系.当母体胎儿方面无异常因素时,胎盘本身的机能是IUGR的重要诱因.作者认为本病并非继发于母体因素,而是由胎盘本身的机能不全所致.为此用电镜观察IUGR胎盘的超微结构.用孕37～42周分娩的,胎儿体重符合IUGR标准,无母体合并症的胎盘13例,并用10例正常产分娩的胎盘作对照.当胎盘娩出后,迅速于靠近脐带附着处采取约1cm大小,肉眼观察无异常的胎盘组织,分别用光镜和电镜观察.检查结果在光镜所见:以甲基绿—派若宁(MGP)染色,甲基绿可使DNA染色,可显示绒毛上皮细胞核内的染色性,正常胎盘和IUGR胎盘之间染色性无显著差别.派若宁可使RNA染色,绒毛上皮细胞的染色性在正常者强,IUGR时低下.胶质铁染色IUGR胎盘染色性低,可推断两种绒毛间质中的酸性粘多糖类的量有差别.电镜所见:在正常胎盘绒毛上皮由合体细胞构成,靠间质的部分有
Intrauterine growth retardation (IUGR) occurs in a complex relationship with maternal, placental fetal function.When there are no abnormal factors in the maternal fetus, the function of the placenta itself is an important incentive for IUGR.The authors believe that the disease is not secondary to maternal factors , But by the placenta itself caused by inadequate performance.To observe the ultrastructure of IUGR placenta by electron microscopy.It was delivered in 37-42 weeks of gestation, fetus body weight consistent with IUGR standards, maternal complications without placenta in 13 cases, and with 10 Cases of normal delivery of the placenta as a control.When the placenta was delivered, quickly near the umbilical cord attached to take the size of about 1cm, no abnormal placental tissue were observed by light and electron microscopy.Results in the light microscope: Methyl Green - Pyrophylline (MGP) staining, methyl green staining DNA can be displayed in the nucleus villus epithelial staining, normal placenta and IUGR placenta between the staining was no significant.Pyronin RNA staining, Chorionic epithelial cells in normal strong staining, IUGR low when the colloidal iron staining IUGR placental staining is low, we can infer the two villous interstitial acid mucopolysaccharide in the amount of the difference. Electron microscopy: in the normal Placental villi epithelium by Somatic constituted by interstitial portion having