鼠感染病毒性肺炎后,肺部的抗菌机制受抑,经常继发细菌感染,多见於病毒感染6—8天之后。在此期间肺泡中的巨噬细胞杀菌活性下降。为了阐明其作用机制,作者用瑞士鼠54只,以吸入感染亚致死量的副流感Ⅰ(仙台)病毒进行实验,并以126只未感染病毒的鼠作对照。二组分开饲养。各於第6 Mouse infected with viral pneumonia, the lungs of the antibacterial mechanism of depression, often secondary to bacterial infection, more common in the virus infection after 6-8 days. During this period the alveolar macrophage bactericidal activity decreased. In order to clarify its mechanism of action, the authors used 54 Swiss mice to infect mice infected with a sufentanil parainfluenza I (Sendai virus), and 126 non-virus infected mice were used as controls. Two groups kept separately. Each at 6th