笔者遇见4例甲胺磷迟发性神经毒性作用之损害,较为少见。为引起临床医生的重视,以寻求预防和有效的治疗措施,兹报道如下: 【例1】韩××,男,18岁,农民,病案号10325,因服甲胺磷原液约20毫升,昏迷1小时,于1984年7月25日下午6时急诊入院。患者平素健康。入院查见:发育营养好,昏迷。呼吸不规则,时有停顿。双瞳孔1毫米。睫毛反射消失。心肺无异常发现,腹壁反射消失,膝腱反射消失。入院诊为重症甲胺磷中毒,经抢救,当晚8时神志转清楚,大汗消失。呼吸规则。瞳孔2毫米。第2病日瞳孔正常。能起床活动,仍诉胸闷。第4病日,患者无不适感,停用阿托 I met four cases of methamidophos delayed neurotoxicity damage, more rare. To attract the attention of clinicians in order to seek prevention and effective treatment measures, it is reported as follows: [Example 1] Han × ×, male, 18 years old, farmer, medical record number 10325, due to taking about 20 ml of methamidophos solution, coma 1 hour, on July 25, 1984 at 6 o’clock emergency admission. Patients usually healthy. See admission: development and nutrition, coma. Irregular breathing, sometimes pause. Double pupil 1 mm. Eyelash reflex disappeared. No abnormal cardiopulmonary findings, abdominal reflex disappeared, knee tendon reflex disappeared. Inpatient diagnosed as severe methamidophos, after the rescue, 8 o’clock that night to clear consciousness, sweating disappeared. Breathing rules. Pupil 2 mm. Day 2 pupil normal. Can get up and still chest tightness. On the 4th day, the patient had no discomfort and disabled Alto