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一氧化氮(NO)是一种活性极强的小分子物质,由一氧化氮合成酶(NOS)催化L-精氨酸合成。适度的NO发挥信号传递作用,过量的NO对组织细胞有损害作用。有报道,力竭游泳大鼠脑组织NOS活性显著增高[1]。海马是脑组织中应激损伤的主要靶区。本实验主要观察了训练大鼠一次性力竭游泳后海
Nitric oxide (NO) is a very active small molecule that catalyzes the synthesis of L-arginine by nitric oxide synthase (NOS). Moderate NO play a role in signaling, excessive NO has a detrimental effect on tissue cells. It has been reported that exhaustive swimming rat brain NOS activity was significantly increased [1]. Hippocampus is the main target of stress injury in brain tissue. The main observation of this experiment training rats after a one-time exhaustive swimming sea