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以马来丝虫接种家猫可引起其后腿淋巴结炎及淋巴管炎并发展为淋巴功能不全.当接种感染后,丝虫便在局部淋巴管中生长.在足部感染24小时内,取自实验室专门培养的蚊虫病媒的丝虫蚴便进入输入淋巴管并游走至淋巴结(腘窝淋巴结)的外周部.丝虫蚴不断地生长,使淋巴管扩张、扭曲、瓣膜功能不全.感染后4—6个月,在这些丝虫中有许多已经分布于足于与腘窝淋巴结之间了.丝虫脱皮、改变寄居部位或在输入淋巴管中死亡时,可发生一系列病理变化.内皮细胞肥大、瓣膜增厚、变形,有时形成淋巴栓子,造成管腔部分性阻塞.管壁中弹力纤维和平滑肌细胞断裂并代之以胶元组织广泛沉着.淋巴管壁内层、淋巴管壁及其周围的结缔组织有炎性细胞积聚.
Inoculation of domestic cats with Malayan filariasis can cause lymphadenitis and lymphangitis in the hind legs and develop into lymphatic insufficiency. When the infection is inoculated, the filarial worms grow in the local lymphatic vessels. Within 24 hours of foot infection, The silkworm larvae, which are specially cultivated by the laboratory, enter the lymphatic vessels and migrate to the periphery of the lymph nodes (popliteal lymph nodes), which continue to grow, causing lymphatic vessels to dilate, distort, and valvular dysfunction. Four to six months after infection, many of these filariasis have been spread between adequate lymph nodes and popliteal lymph nodes. A series of pathological changes may occur when filarial peels, alter the domicile, or die in the input lymphatic vessels Endothelial cell hypertrophy, valve thickening, deformation, and sometimes the formation of lymphatic emboli, resulting in partial obstruction of the lumen.Elastic fibers and smooth muscle cells in the wall of the fracture and replaced by plastic collagen tissue extensive deposition.Limbic wall lining, lymph Inflammatory cells accumulate in the connective tissue around the wall and around it.