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测试了12个致病力不同的Xanthomonas oryzae Pv.oryzae 菌株,其EPS产生能力受培养周期和可利用糖的影响。多糖只有1个级分,不含蛋白成分,分子最小于2×10~6道尔顿。其非专化性毒性包括堵塞植物幼苗切条导管而致萎,导致被注有EPS的植物组织变裼坏死;诱使植细物胞电解质渗漏。此外,弱致病力菌株的FPS具有诱导品种对白枯病的抗性能力。初步认为,白叶枯病菌EPS可能与症状形成有关,并参与寄主一病菌相互作用过程中对水稻外源凝集素的识别,但EPS可能只是病菌致病力的构成之一。
Twelve virulent strains of Xanthomonas oryzae Pv.oryzae were tested and their EPS production ability was influenced by the culture cycle and available sugar. Polysaccharides only one fraction, excluding protein components, the smallest molecule in 2 × 10 ~ 6 Dalton. Its non-specific toxicity includes plugging the seedlings of cutting pipe and lead to wilt, resulting in plant tissue infiltrated with EPS become necrotic; inducing phytoplankton electrolyte leakage. In addition, FPS of weak pathogenic strains have the ability of inducing varieties to resist bacterial wilt. It is preliminarily believed that EPS may be involved in the formation of symptoms and may be involved in the identification of rice lectin during the interaction of host-host bacteria, but EPS may only be one of the pathogenic components of pathogenic bacteria.