论文部分内容阅读
研究者们曾报告慢性活动性肝炎(简称慢活肝)的形成可以是遗传因素或是接触病毒或药物而引起的。本文作者研究了这两种类型慢活肝的病程和结局。将45例研究对象分成三组:第Ⅰ、Ⅱ组是由外源性因子诱发的慢活肝25例(由病毒诱发的13例、药物诱发的12例),第Ⅲ组是隐匿型的慢活肝20例。此外,健康对照者690例。作者用淋巴毒素微量技术对研究对象进行 HLA 型的检测,同时,也进行肝功能、血清内丙种球蛋白和自身抗体的检测以及肝活检。结果如下:
Researchers have reported that the formation of chronic active hepatitis (referred to as slow living liver) can be caused by genetic factors or exposure to viruses or drugs. The authors studied the course and outcome of these two types of slow-acting liver. Forty-five subjects were divided into three groups: group I and group II were 25 cases of slow-acting liver induced by exogenous factors (13 cases induced by virus and 12 cases induced by drugs), and group III was occult slow Live liver in 20 cases. In addition, 690 healthy controls. The author used lymphotoxin micro-technology to carry out the detection of HLA type in the study subjects, meanwhile, he also performed liver function tests, detection of serum gamma-globulin and autoantibodies and liver biopsy. The result is as follows: