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作者首次通过对大鼠急性坏死性胰腺炎(ANP)模型进行了肝脏能量代谢的研究,发现在ANP早期,肝脏通过线粒体氧化磷酸化活性增强来增加ATP的合成,使肝脏能荷尚能维持于接近正常的水
The authors first studied the hepatic energy metabolism in rats with acute necrotizing pancreatitis (ANP) model and found that in the early stage of ANP, the liver increased the synthesis of ATP through enhanced mitochondrial oxidative phosphorylation activity, Near normal water