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急性病毒性肝炎的潜伏期或早期中,出现一种白蛋白相关的亲脂分子,经验明并确定是血清抑制因子。这种抑制因子可干扰静止的 T 淋巴细胞和 B 淋巴细胞,但不能干扰已活化的 T 淋巴细胞和B 淋巴细胞。很多作者已报告,急性病毒性肝炎完全痊愈和血中 SIF 消失具有平行的相互关系,血中 SIF 持续存在,乃表示肝炎病毒仍在进行复制。为了研究上述发现的临床可靠性,本文作者对10例急性甲型、58例急性乙型和13例急性非甲非乙型病毒性肝炎共81例进行临床观察一年,在这一年中,于疾病的第1周以及病后4、8、24和52周测定生化学、血清学的指标与 SIF 活性,以
During the incubation or early stages of acute viral hepatitis, an albumin-related lipophilic molecule appears that has been identified and identified as a serum inhibitory factor. This inhibitor interferes with resting T lymphocytes and B lymphocytes but does not interfere with activated T lymphocytes and B lymphocytes. Many authors have reported a parallel relationship between the complete recovery of acute viral hepatitis and the disappearance of SIF in blood, and the persistence of SIF in blood, indicating that hepatitis virus is still being replicated. In order to study the clinical reliability of the above findings, the authors of the clinical trial of 10 cases of acute A, 58 cases of acute B and 13 cases of non-A non-B hepatitis A clinical observation for one year, during this year, Biochemical, serological markers and SIF activity were measured at week 1 of disease and at 4, 8, 24 and 52 weeks after illness to