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本实验以人工外淋巴灌流方式，提高豚鼠耳蜗外淋巴液钙离子浓度（［Ｃａ２＋］ＰＬ），观察蜗内直流电位（ＥＰ）和耳蜗电图（ＥＣｏｃｈＧ）的变化，ＥＣｏｃｈＧ包括听神经复合动作电位（ＣＡＰ）、耳蜗微音电位（ＣＭ）。结果可见：高钙灌流明显抑制ＣＡＰ幅值，延长同一声强下（９０ｄＢＳＰＬ）Ｎ１－峰潜伏期，但不改变ＣＭ的幅值及总和ＥＰ（Ｇ－ＥＰ）。高钙灌流降低了ＥＰ对噪声的给－撤声反应（ＥＰ－ＯＮ，ＥＰ－ＯＦＦ）和缺氧所得到的最大负ＥＰ（Ｎ－ＥＰ）绝对值。本文分析了外淋巴高钙影响耳蜗电位的可能机制。
In this experiment, the extracorporeal lymphatic fluid calcium concentration ([Ca2 +] PL) in guinea pigs was increased by artificial perilymph perfusion to observe the changes of DC and ECochG contents in the cochlea. ECochG includes the auditory nerve complex action potentials CAP), cochlear microphones (CM). The results showed that: high calcium perfusion significantly inhibited the amplitude of CAP and prolonged the N1-peak latency under the same sound intensity (90dBSPL), but did not change the amplitude of CM and the total EP (G-EP). High-calcium perfusion decreased the EP-ON response (EP-ON) and the absolute maximum negative EP (N-EP) value obtained from hypoxia. This article analyzes the possibility of hypermetabolism of cochlear implants.