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一氧化碳是一种无色、无味、无刺激性的窒息性气体,机体短时间内吸入可引起急性中毒。一氧化碳中毒主要引起组织缺氧,可致脑血栓形成、脑皮质和基底节局灶性的缺血性坏死以及广泛的脱髓鞘病变,如果不能得到及时有效的治疗,部分患者会留有后遗症,出现精神意识障碍、震颤麻痹综合征、偏瘫、病理反射阳性、失语、失明、继发性癫痫、脑神经及周围神经损害等,即迟发性脑病。国外报道急性一氧化碳中毒患者出现一氧化碳中毒后迟发性脑病的比例高达3%~40%[1]。高压
Carbon monoxide is a colorless, odorless, non-irritating asphyxiant gas that can cause acute poisoning when inhaled for a short period of time. Carbon monoxide poisoning is mainly caused by tissue hypoxia, can cause cerebral thrombosis, cerebral ischemic necrosis of the cortex and basal ganglia and extensive demyelinating disease, if not be timely and effective treatment, some patients will have sequelae, Appear mentally disturbed, paralysis paralysis syndrome, hemiplegia, pathological reflex positive, aphasia, blindness, secondary epilepsy, cranial nerve and peripheral nerve damage, that is, delayed encephalopathy. Foreign reports of acute carbon monoxide poisoning in patients with carbon monoxide poisoning delayed encephalopathy up to 3% to 40% [1]. high pressure