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本文观察了14例无氮质血症糖尿病患者磷负荷4周前后的尿蛋白变化。发现磷负荷使血糖控制者尿蛋白排泄增加,血糖未控制者通过磷负荷加常规治疗或单纯常规治疗改善血糖后尿蛋白均减少。血糖控制者经磷负荷尿磷净增值较血糖未控制者高70％。结果提示磷摄入过量具有肾毒性作用,显著高尿磷可能参与糖尿病患者蛋白尿的形成。文中就此探讨了合理补磷治疗糖尿病的问题。
In this paper, we observed the changes of urinary protein in 14 patients with no azotemia before and after phosphorus loading for 4 weeks. Found that phosphorus load increased blood glucose control urinary protein excretion, blood glucose control by phosphorus load plus conventional therapy or simple routine therapy to improve blood sugar decreased urinary protein. Blood glucose control by the phosphorus load of urine phosphorus increased more than 70% of the uncontrolled blood glucose. The results suggest that excessive phosphorus intake has a nephrotoxic effect, significantly higher urinary phosphorus may be involved in the formation of proteinuria in diabetic patients. In this paper, to explore the reasonable phosphorus for the treatment of diabetes problems.