给大鼠反复ipCCl4 诱发肝脏代谢紊乱 ,脂质沉积 ,代偿性地使肝内弥漫性纤维增生 ,肝脏假小叶形成 ,肝组织中HPA含量增加 ,形成肝硬化 ,并可见血清ALT活性升高 ,TP与ALB减少 ,A/G比值倒置。肝复乐能降低CCl4 肝硬化大鼠模型血清和肝组织中TCh和TG含量 ,升高血清TP与ALB ,逆转A/G比值 ,降低ALT活性 ,降低肝组织中HPA含量。表明肝复乐对CCl4肝硬化具有保护作用 Rats with repeated ipCCl4 induced hepatic metabolic disorders, lipid deposition, compensatory intrahepatic diffuse fibrosis, hepatic pseudolobule formation, increased hepatic tissue HPA levels, cirrhosis, and elevated serum ALT activity. The TP and ALB are reduced, and the A/G ratio is inverted. It can reduce the serum and hepatic TCh and TG levels in CCl4 liver cirrhosis rat models, increase serum TP and ALB, reverse the A/G ratio, reduce ALT activity, and reduce hepatic tissue HPA levels. Shows that Ganfele has a protective effect against CCl4 liver cirrhosis