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为了解先天性肛门直肠畸形的病理改变，用乙烯硫脲（ＥＴＵ）诱发大白鼠产生肛门闭锁畸形鼠仔，观察其盆腔病理改变。结果：低位肛门闭锁肛门内括约肌正常；高、中位有瘘型肛门闭锁存在内括约肌，但直肠前壁短小，畸形位置越低，内括约肌面积越大；高、中位无瘘型肛门闭锁无内括约肌。有瘘型肛门闭锁及低位肛门闭锁直肠远端或瘘管内覆未角化的复层上皮或角化的鳞状上皮，为正常肛管的异位。肛门闭锁畸形鼠仔外括约肌缺如、异位或正常，肛提肌薄弱。肛门闭锁畸形术中应充分保留瘘管近端及直肠盲端组织。
In order to understand the pathological changes of congenital anorectal malformations, rats with anal atresia were induced by ethylene thiourea (ETU) to observe the pathological changes of the pelvis. Results: The internal anal sphincter was normal in the low anus atresia. The internal sphincter was found in the high and middle fistula anus. However, the anterior wall of the rectum was short and the position of the anterior rectum was lower. The internal sphincter area was larger. The high and medium fistula-free anal atresia Internal sphincter. There are fistula anus atresia and low anorectal atresia remote rectal fistula or non-keratinized stratified epithelial or keratinized squamous epithelium, the normal anal canal ectopic. Anal atresia Aberrant sphincter absent, ectopic or normal, levator ani muscle weak. Anal atresia surgery should be fully retained fistula proximal and rectum blind end of the organization.