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以颌下腺为实验模型观察交感神经损伤后副交感神经可能发生的改变,现在已从两方面取得了进展。一是用生化方法测定颌下腺内胆碱乙酰化酶(ChAT),发现去交感神经或化学断交感纤维后该酶活力明显增高;二是通过组化技术进行的形态学定性和半定量研究,观察到去交感神经后溃变的颌下交感干内有乙酰胆碱酯酶(AChE)阳性纤维长入,切断交感节前纤维亦可引起腺实质内AChE阳性纤维末梢密度有所增加。上述研究均提示支配颌下腺的交感神经损伤将导致副交感神经发生某些代偿性反应。本实验用放射免疫法测定了交感神经损伤后大鼠颌下腺中胆碱能神经递质——乙酰胆碱(ACh)浓度的变化,并观察了断交感节
Taking submandibular gland as experimental model to observe the possible changes of parasympathetic nerve after sympathetic nerve injury, we have made some progress in two aspects. First, the use of biochemical methods for determination of subchyamal choline acetyltransferase (ChAT), found that after the sympathetic nerve or chemical broken off the fiber significantly increased the activity of the enzyme; the second is through the histochemical qualitative and semi-quantitative studies were observed AChE-positive fibers grew in the submandibular sympathetic trunk after degeneration of the sympathetic nerve, and cutting off the sympathetic preganglionic fibers also caused an increase in the density of AChE-positive terminal fibers in the glandular parenchyma. The above studies all suggest that the sympathetic nerve damage that governs the submandibular gland will result in some compensatory responses to the parasympathetic nerve. In this experiment, the change of cholinergic neurotransmitter-acetylcholine (ACh) concentration in rat submandibular gland after sympathectomy was measured by radioimmunoassay,