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目的初步探讨抑郁障碍对颅脑创伤患者反应抑制功能的影响。方法采用Glasgow昏迷量表、汉密尔顿抑郁量表17项(HAMD-17)和日常生活活动能力量表(ADL)评价104例颅脑创伤患者(伴抑郁障碍54例、不伴抑郁障碍50例)颅脑创伤和抑郁障碍严重程度,刺激-反应相容性试验记录反应抑制任务反应时间。结果颅脑创伤伴抑郁障碍组HAMD-17(P=0.000,0.000)和ADL(P=0.000,0.000)评分高于颅脑创伤不伴抑郁障碍组和对照组,颅脑创伤不伴抑郁障碍组HAMD-17(P=0.000)和ADL(P=0.000)评分亦高于对照组。无论执行相容性还是不相容性任务,颅脑创伤伴或不伴抑郁障碍组患者反应时间均长于对照组(P=0.000,0.000),颅脑创伤伴抑郁障碍组患者反应时间亦长于颅脑创伤不伴抑郁障碍组(P=0.000)。结论颅脑创伤后可发生认知功能障碍,且在伤后6个月或更长时间仍存在。颅脑创伤伴抑郁障碍可以加重患者认知功能障碍,应早期识别并及时干预。
Objective To investigate the effect of depressive disorder on response inhibition in patients with traumatic brain injury. Methods A total of 104 patients with craniocerebral trauma (54 with depressive disorder and 50 without depressive disorder) were evaluated using the Glasgow Coma Scale, Hamilton Depression Rating Scale 17 (HAMD-17) and the Living Activities Accomplishment Scale (ADL) Brain trauma and depressive disorder severity, Stimulus - Response Compatibility Test recorded responses to suppress task response time. Results The scores of HAMD-17 (P = 0.000,0.000) and ADL (P = 0.000,0.000) in patients with craniocerebral trauma and depressive disorder were significantly higher than those in patients with traumatic brain injury without depressive disorder and control group, with traumatic brain injury without depressive disorder The scores of HAMD-17 (P = 0.000) and ADL (P = 0.000) were also higher than those of the control group. The response time of patients with craniocerebral trauma or without depressive disorder was longer than that of the control group (P = 0.000,0.000), no matter whether they performed compatibility or incompatibility tasks. Patients with craniocerebral trauma and depressive disorder also had longer reaction time Brain trauma was not associated with depression (P = 0.000). Conclusion Cranial trauma can occur after cognitive impairment, and still exist after 6 months or more. Craniocerebral trauma with depression can aggravate cognitive dysfunction in patients and should be recognized early and timely intervention.