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注射大量肾上腺素后,变性心肌纤维可以呈现异常糖原的堆聚,脂肪变性及硫氢基增多现象,这是心肌受损的表征。这种受损的纤维可以康复,亦可进一步的变性,以致坏死,形成病灶。病灶周围的心肌亦呈上述损伤表征。单一次注射大量肾上腺素14日后,病灶已结瘢修复,瘢疤周围心肌基本正常,仅极少数纤维仍呈变性现象。本文着重讨论了在受损的心肌内,脂肪变性及异常糖原二者间的代谢关系,同时也探讨了蛋白质变性对脂肪及糖原二者代谢的可能影响。由于肾上腺素对心肌的影响,大量使用时应谨妥为宜。
After injection of a large amount of epinephrine, degenerative myocardial fibers can exhibit abnormal glycogen accumulation, steatosis and sulfhydryl groups, which is a sign of myocardial damage. This damaged fiber can be recovered, but also further degeneration, resulting in necrosis, the formation of lesions. The myocardium around the lesion was also characterized by the above lesions. A single injection of large amounts of epinephrine 14 days after the lesion has knot scar repair, myocardium around the scar is normal, only a very small number of fibers are still denaturing phenomenon. This article focuses on the metabolic relationship between steatosis and abnormal glycogen in impaired myocardium and the possible effects of protein denaturation on the metabolism of both fat and glycogen. Due to the impact of epinephrine on the myocardium, should be appropriate when used in large quantities.