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室内对峙培养和寄生致腐作用测定结果表明 :盾壳霉 (ConiothyriumminitansCampbell)对核盘菌菌丝生长没有明显拮抗作用 ,但对菌核形成有一定抑制作用。被盾壳霉寄生的菌核表面皱缩、软化腐烂 ,后期菌核表面密生大量盾壳霉分生孢子器 ,并从顶端孔口分泌出黑色球状分生孢子黏液。显微镜检盾壳霉寄生的腐烂菌核石蜡切片 ,结果表明 :盾壳霉菌可在菌核的表面和内部疏丝组织寄生 ,并形成分生孢子器。盾壳霉分生孢子器表生、半埋生或埋生于腐烂菌核上。被盾壳霉寄生的菌核 ,后期拟薄壁组织消解断裂成不连续的残片 ,内部疏丝组织消解 ,在发病后期致密的疏丝组织被盾壳霉菌消解成的网状结构所代替。
Laboratory confrontation culturing and parasitization decay results showed that: Coniothyrium minitans Campbell had no obvious antagonism on the mycelial growth of Sclerotinia sclerotiorum, but had some inhibition on the formation of sclerotia. Colletotrichum parasitic sclerotinia surface shrinkage, softening and decay, the late sclerotia surface dense conidia pyrenoidosa conidia, and from the top of the hole to secrete black spherical conidia mucus. Microscopic examination of Trichoderma parasitic rot fungus nuclear paraffin section, the results show that: Sclerotinia mold in the sclerotium on the surface and internal sparse tissue parasitic silk, and the formation of conidia. Conch shell conidia superficial, half buried or buried in the rot of bacteria. Bacillus subtilis parasitic sclerotia, the latter part of the proposed parenchyma tissue digestion broken into pieces of non-continuous, internal sparse tissue digestion, in the late onset of dense sparse tissue was replaced by Tricholomaceae disintegration into the network structure.