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实验在15只家兔上进行。以玻璃微电极记录尾核头部(DHCN)痛相关单位,刺激扣带回,观察痛相关单位电活动的变化。在70只DHCN痛相关单位中,有44个对刺激扣带回表现兴奋反应(62.9％),17个呈抑制反应(24.3％),9个变化不明显(12.8％)。进一步观察了52只痛相关单位,在刺激扣带回后再施伤害刺激,43个原痛兴奋单位中15个转为抑制(34.9％),16个进一步兴奋(37.2％)12个无变化(27.9％);原9个痛抑制单位,8个转为兴奋,1个变化不明显。提示,扣带回可通过其下行活动,调制伤害性冲动在尾核水平上的传递。
Experiments were performed on 15 rabbits. The glassy microelectrode was used to record the related units of pain in the caudate nucleus head (DHCN), stimulating the cingulate gyrus, and observing the changes of electrical activity in pain-related units. Of the 70 DHCN-related units, 44 showed an excitatory response to stimulus cingulate reflux (62.9%), 17 showed an inhibitory response (24.3%), and 9 changed insignificantly (12.8%). Fifty-two pain-related units were further observed, followed by stimulation of excitability after stimulation of the cingulate gyrus. Fifteen of the 43 patients with primitive pain were converted to inhibition (34.9%) and 16 to be further excited (37.2%), 12 unchanged 27.9%); the original nine pain inhibition units, eight to excited, a change is not obvious. Tip, Cingulate back through its activities down, modulation of harmful impulses at the level of the caudate nucleus transmission.