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近期的一些研究证实，吸入低浓度一氧化氛（NO）有扩张肺血管的作用。考虑到CO和NO的理化性质相似，因此我们设想低浓度的CO对大鼠成体肺血管亦有和NO相同的作用。本研究用雄性大鼠高体的经盐水灌注的具有正常肺动脉压（CON）和肺动脉高压（ALT）的肺脏．以确定CO（200和1000PP
Some recent studies have confirmed that inhaled low concentrations of nitric oxide (NO) have the role of pulmonary vasodilation. Considering the similar physical and chemical properties of CO and NO, we hypothesize that low concentrations of CO also have the same effect as NO on adult pulmonary blood vessels in rats. In this study, lungs with normal pulmonary arterial pressure (CON) and pulmonary hypertension (ALT) were perfused with saline in male rats. To determine the CO (200 and 1000PP