热休克是以暴露于高温潮湿气候、高热和神经系统异常为特征的疾病,是热带及温带地区热浪期间的主要致病和致死原因。从暴露于高温发展到高热,以及在一些情况下发展到多器官功能衰竭和死亡的机制还不十分清楚。很多资料提示,内毒素血症和细胞因子在致病机制中起了作用。本研究旨在:①确定热休克时可溶性肿瘤坏死因子受体(sTNFR60、sTNFR80) Heat shock is a disease characterized by exposure to hot and humid climates, hyperthermia and neurological abnormalities and is the leading cause of disease and death during heat waves in tropical and temperate regions. The mechanism of development from hyperthermia to hyperthermia, and in some cases to multiple organ failure and death, is not well understood. Many sources suggest that endotoxemia and cytokines play a role in the pathogenesis. The purpose of this study was to: (1) To determine the expression of soluble tumor necrosis factor receptor (sTNFR60, sTNFR80)