因顿伤或挫伤导致房角后退续发青光眼,早在1881年 Smith 等人进行了研究,国内也有报告。Blanton 统计130例房角后退病例,7%发生青光眼。有二个发病率较高的时期:约11%挫伤病人在外伤后3年内发病。挫伤初期房水分泌功能受抑制,往往还有房水流出阻力增大,外伤后3～36个月,随着房水分泌功能逐渐恢复,眼压升高,症状明显化;也有滤帘功能恢复,房水流出阻力恢复正常。另一时期约20%的患者,在外伤后 Glaucoma was caused by retrograde angle retrogression due to injury or contusion. As early as 1881, Smith et al. Conducted studies and reports were also made in China. Blanton statistics retrospective cases of 130 cases, 7% of glaucoma. There were two periods of high morbidity: About 11% of bruising patients developed disease within 3 years after trauma. Precontract aqueous humor secretion was inhibited, there is often room outflow resistance increases, 3 to 36 months after trauma, with the recovery of aqueous humor gradually restored, intraocular pressure increased, the symptoms clear; there are also filter curtain function recovery , Atrial outflow resistance returned to normal. About 20% of patients in another period after trauma