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目的:探讨癌症发病机理。方法应用同位素掺入法、酶联法检测了癌症患者血浆白细胞介素1(IL-1)活性和肿瘤坏死因子(TNF)含量。结果:癌症患者血浆IL-1活性显著低于对照组,而血浆TNF含量却显著高于对照组。结论:肿瘤患者巨噬细胞活性抑制,TNF可能部分来自肿瘤细胞自分泌。
Objective: To investigate the pathogenesis of cancer. Methods The levels of plasma interleukin-1 (IL-1) and tumor necrosis factor (TNF) in cancer patients were detected by isotope incorporation and enzyme-linked immunosorbent assay. Results: The plasma IL-1 activity in cancer patients was significantly lower than that in the control group, but the plasma TNF level was significantly higher than that in the control group. Conclusion: The activity of macrophages in tumor patients is inhibited, and TNF may be partly from autocrine of tumor cells.