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目前研究发现在心肌梗塞时,由于缺血、缺氧所致受损心肌组织,在其破坏过程中,可长时间地,不断地向血中释放心肌肌球蛋白轻链。心肌梗塞后,血中轻链值的时相性变化与心肌肌原纤维破坏的时相性经过基本一致,故轻链值的时相性变化在反映肌原纤维破坏与修复的过程上具有意义。此外,在实验性心肌梗塞研究中,利用轻链值达高峰时间晚,且上升持续时间长的特点,比用CPK为主的酶测定指标来推测心肌损害的进展情况更为优越。
The current study found that myocardial infarction, due to ischemia, hypoxia caused by damage to myocardial tissue, in the process of its destruction, long time and continuously to the release of myocardial myosin light chain. Myocardial infarction, the blood light chain value of the phase changes and myocardial myofibrillar destruction of the phase after basically the same, so the light chain value of the phase changes in the reflection of myofibrillar destruction and repair of significance. In addition, in the experimental myocardial infarction study, the use of light chain value of the peak late, and the long duration of the rise of the characteristics, than the CPK-based enzyme assay to predict the progress of myocardial damage is more superior.