乙胺碘呋酮诱发甲状腺毒症(AIT)常发生于居住在碘摄入不足地区的病人中,而环境中碘摄入量正常时更常发生甲状腺功能低下症。一般认为 AIT 是由于乙胺碘呋酮脱碘作用释放出碘化物而导致甲状腺激素过量产生和释放,恃别是在具有甲状腺功能正常的结节性自律甲状腺和 Graves’氏病患者中是这样。近年来有人提出 AIT 可能是由于甲状腺内的一个炎症病程,导致过量甲状腺激素释放入血循环系统所致。这一过程与在具 Amiodarone-induced thyrotoxicosis (AIT) often occurs in patients who live in regions with inadequate iodine intake, and hypothyroidism is more common when iodine intake is normal in the environment. It is generally believed that AIT is caused by the iodine deficiency of amiodarone to release iodine, which leads to overproduction and release of thyroid hormone. This is the case in patients with nodular autonomic thyroid and Graves’ disease who have normal thyroid function. In recent years, it was suggested that AIT may be due to an inflammatory process in the thyroid gland, resulting in the release of excess thyroid hormone into the circulatory system. This process is in progress