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自1963年在乙型肝炎患者的血清中发现乙型肝炎表面抗原(HBsAg)以来,曾在唾液,精液,乳汁,胆汁,肠腔积液,腹水,尿及粪内检出。从而使对乙型肝炎传播途径有了进一步的认识。现将我院近年来检出的2例腹水(HBsAg)阳性病人报告如下: 例1:男,39岁。住院号1090。患者于1970年患急性乙型肝炎,迁延不愈,症状逐年加重。1984年2月因病情加重,收住我院,诊断为肝炎后肝硬化,检查:呈肝病面容,巩膜黄染,肝掌,腹部膨隆,有移动性浊音,肝未扪及,脾肋下6厘米。肝功能异常,HBsAg阳性。腹水报告:淡黄,微混,比重1.012,利凡他阴性,白细胞60,HBsAg阳性。例2:男,18岁,住院号9758。患者1981年初患急性黄疸型肝炎,住某院,血清中检出HBsAg阳性,
Since 1963, hepatitis B surface antigen (HBsAg) was found in the serum of hepatitis B patients, saliva, semen, milk, bile, intestinal effusion, ascites, urine and feces were detected. So that the spread of hepatitis B have been further understanding. Now in our hospital in recent years detected 2 cases of ascites (HBsAg) -positive patients reported as follows: Example 1: Male, 39 years old. Hospital number 1090. Patients with acute hepatitis B in 1970, delayed healing, the symptoms increased year by year. February 1984 due to aggravating condition, admitted to our hospital, diagnosed with hepatitis cirrhosis, check: liver disease face, scleral yellow dye, liver palms, abdominal bulging, with shifting dullness, liver palpable, spleen ribs 6 cm. Abnormal liver function, HBsAg positive. Ascites report: light yellow, slightly mixed, the proportion of 1.012, Lee Van He negative, white blood cells 60, HBsAg positive. Example 2: Male, 18 years old, hospital number 9758. Patients with acute jaundice hepatitis in early 1981, living in a hospital, HBsAg positive serum detected,