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为明确黄斑囊样水肿(CME)的病理改变,本文研究了35例具有CME组织病理学改变的人眼,发现在白内障摘出术后、视网膜静脉阻塞、外伤、糖尿病和进行性高血压,其囊可出现在视网膜的不同层次内。病理过程包括漏出、渗出和液化性坏死。本文并观察了用恒河猴做的晶体摘出和滑石粉视网膜病变的模型。在晶体摘出后发现血-视网膜屏障在视网膜血管和视网膜色素上皮的部位遭到破坏。在猴滑石粉视网膜病变中见有黄斑囊样变性。根据以上结果作者提出:在CME形成过程中,血-视网膜屏障的破坏和视网膜微小梗塞可能起重要作用。黄斑囊样水肿(CME)可发生于多种病理情况下,如在糖尿病性视网膜病变、视网膜中央静脉或分枝阻塞、高血压性视网膜病变和其他一些营养性和代谢性疾病以及白内障摘出术、视网膜脱
In order to clarify the pathological changes of cystoid macular edema (CME), 35 human eyes with histopathological changes of CME were studied in this paper. After cataract extraction, retinal vein occlusion, trauma, diabetes and progressive hypertension were observed. Can appear in different levels of the retina. Pathological processes include leakage, exudation and liquefaction necrosis. This article also examines the use of rhesus monkeys extracted and talc model of retinopathy. After the crystal was removed, the blood-retinal barrier was found to be damaged in the retinal blood vessels and the retinal pigment epithelium. Cystoid macular degeneration was seen in the monkey talc retinopathy. According to the above results, the authors suggest that the destruction of the blood-retinal barrier and the subtle infarction of the retina may play an important role in the formation of CME. Macular cyst edema (CME) can occur in a variety of pathological conditions, such as diabetic retinopathy, central retinal vein or branch occlusion, hypertensive retinopathy and other nutritional and metabolic diseases and cataract extraction, Retinal detachment