从分子和生化基础上来了解感染性休克综合征仍然很少。细菌产物与人的相互作用有两个重要机理:第一是对真核细胞的直接作用,引起从细胞激活到细胞死亡和溶解的反应;第二是正常机体防卫系统产物所介导的间接作用,最终也引起类似的反应,即从细胞的激活至死亡。 From the molecular and biochemical basis to understand septic shock syndrome is still very small. There are two important mechanisms by which bacterial products interact with humans: the first is a direct effect on eukaryotic cells, causing a response from cell activation to cell death and lysis; the second is an indirect effect mediated by normal body defense system products , Eventually led to a similar reaction, from the activation of cells to death.