成人呼吸窘迫综合症(ARDS)是各科危重病常见的并发症。它的病理改变主要是肺微血管内皮细胞及肺泡上皮细胞的损害,由于微血管内皮细胞的损坏,而间质水肿间质腔内压力增加,继之肺泡水肿,表面活性物质失活,因而出现弥漫性肺泡不张,还可能同时存在肺微循环障碍,在此基础上,导致肺内分流及肺泡死腔增加,功能残气量减少,肺顺应性降低,严重低氧血症等病理生理变化。要对该症病 Adult respiratory distress syndrome (ARDS) is a common complication of various critical illness. Its pathological changes are mainly the damage of pulmonary microvascular endothelial cells and alveolar epithelial cells, due to the damage of microvascular endothelial cells, interstitial edema interstitial cavity pressure increased, followed by alveolar edema, surfactant inactivation, resulting in diffuse Alveolar atelectasis, pulmonary microcirculation may also exist at the same time, on this basis, leading to pulmonary shunt and increased dead space, decreased functional residual capacity, decreased lung compliance, severe hypoxemia and other pathophysiological changes. To the disease