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C—反应蛋白因能与肺炎球菌多糖体C起反应后与之形成沉淀而得名。 C—反应蛋白在健康人不存在,而在炎症后坏死組織的許多病理情况下才出現。Tilletr与Francis确定肺炎有C—反应蛋白,Anderson与Mccarty諸氏确定风湿症亦有此蛋白,Lofstrom最早指出心肌梗死患者有C—反应蛋白,对其引起肺炎球菌莢膜肿涨的C—反应蛋白与物貭的同一性被証实。 Phear与Sfirland曾发現15例心肌梗死患者中,14例有C—反应蛋白,Kroop等覌察32例心肌梗死患者均有此蛋白,Levinger从50例中发現49例,Wager等94％病例中有之。作者应用軍医学院敎研室氏制作的免疫血清,并应用他所更改的Anderson与Mc Carty氏方法測定C—反应蛋白,沉淀小柱按毫米計。按其高度鑑定蛋白量。(亦依照法)。无沉淀为阴性反应,沉渣小于1毫米为痕迹,沉渣在1毫米为弱阳性,2—3毫米为阳性,沉渣在4毫米以上为强阳性。作者在52例心肌梗死,44例狭心症患者中研究了这种反应。
C-reactive protein due to pneumococcal polysaccharide C react with the formation of a precipitate after its name. C-reactive protein does not occur in healthy people, but occurs in many pathological conditions of necrotic tissue after inflammation. Tilletr and Francis to determine pneumonia with C-reactive protein, Anderson and Mccarty Zhu confirmed that rheumatoid disease also has this protein, Lofstrom first pointed out that patients with myocardial infarction C-reactive protein, causing pneumococcal capsule swelling C-reactive protein and The identity of the object is confirmed. Phear and Sfirland have found 15 patients with myocardial infarction, 14 patients with C-reactive protein, Kroop et al observed 32 patients with myocardial infarction patients with this protein, Levinger found in 50 cases of 49 cases, Wager and other 94% of cases There are in it. The authors applied the immune sera produced by the Institute of Pharmacology and Pharmacology, and used his modified Anderson and Mc Carty’s method to measure C-reactive protein. The pellet was measured in millimeters. According to their height identification of protein. (Also according to law). No sediment negative reaction, less than 1 mm sediment marks, sediment weak positive at 1 mm, 2-3 mm positive, more than 4 mm sediment is strong positive. The authors studied this response in 52 patients with myocardial infarction and 44 patients with angina pectoris.