论文部分内容阅读
目的探讨急性胰腺炎腹膜后隙的CT炎性浸润程度与临床严重程度的相关性。资料与方法回顾性分析118例急性胰腺炎CT表现,观察腹膜后隙的炎性浸润程度,与急性胰腺炎临床严重程度进行对比分析。结果118例中,98例不同程度累及腹膜后隙,阳性率83.1%(98/118),肾旁前间隙、肾周间隙、肾旁后间隙总的受累率分别为83.1%(98/118)、62.7%(74/118)、31.4%(37/118)。其中轻症急性胰腺炎44例,主要累及肾旁前间隙,腹膜后隙炎性浸润程度以0级和Ⅰ级为主,分别占45.5%(20/44)和47.7%(21/44);重1型急性胰腺炎33例,同时累及肾旁前间隙和肾周间隙,炎性浸润程度以Ⅱ级为主,占78.8%(26/33);重2型急性胰腺炎41例,同时累及肾旁前间隙、肾周间隙和肾旁后间隙,炎性浸润程度均在Ⅱ级以上,并以Ⅲ级为主,占80.5%(33/41)。腹膜后隙的炎性浸润程度与临床急性胰腺炎的严重程度呈显著正相关(r=0.7797,P=0.000)。结论腹膜后隙的CT炎性浸润程度,特别是肾旁后间隙的受累往往反映了急性胰腺炎的临床严重程度。
Objective To investigate the correlation between CT inflammatory infiltration and clinical severity in retroperitoneum of acute pancreatitis. Materials and Methods Retrospective analysis of 118 cases of acute pancreatitis CT manifestations, the degree of inflammatory infiltration of the retroperitoneum was observed and compared with the clinical severity of acute pancreatitis. Results Of the 118 cases, 98 cases of retroperitoneum were involved in varying degrees, with a positive rate of 83.1% (98/118). The total pre-renal peritoneal space, perirenal space, and posterior paraventricular space were 83.1% (98/118) , 62.7% (74/118), 31.4% (37/118). Forty - four cases with mild acute pancreatitis mainly involved the anteroepithelial space. The degree of peritoneal inflammatory infiltration was grade 0 and grade Ⅰ, accounting for 45.5% (20/44) and 47.7% (21/44), respectively. Severe type 1 acute pancreatitis in 33 cases, both involved the anterior renal peritoneal space and perirenal space, the degree of inflammatory infiltration mainly in grade Ⅱ, accounting for 78.8% (26/33); severe acute pancreatitis type 2 in 41 cases, involving both The anterolateral anterior renal space, the perirenal space, the posterior renal access space, and the inflamed infiltration were all above grade Ⅱ, and were mainly grade Ⅲ, accounting for 80.5% (33/41). The degree of inflammatory infiltration in the retroperitoneum was positively correlated with the severity of clinical acute pancreatitis (r = 0.7797, P = 0.000). Conclusion Peritoneal clearance CT inflammatory infiltration, especially the involvement of the posterior renal access space often reflects the clinical severity of acute pancreatitis.