2. 您的位置
3. 首页
4. 会议论文
5. RECURRENT SYNCOPE AS INITIAL SYMPTOM IN PHARYNX SQUAMOUS CELL CARCINOMA

RECURRENT SYNCOPE AS INITIAL SYMPTOM IN PHARYNX SQUAMOUS CELL CARCINOMA

来源 :第二十四届长城国际心脏病学会议、亚太心脏大会暨国际心血管病预防与康复会议(The 24th Great Wall Int | 被引量 : 0次 | 上传用户：kevinsnower

【摘 要】

：

Objectives Syncope is caused by a wide variety of disorders.Recurrent syncope as a complication of malignancy is uncommon and may be difficult to diagnose and to treat.Methods In such cases, surgery,

【作 者】

：

Chen Jian Fei Huanglan 

【机 构】

：

Department of Cardiology,The Second Affiliated Hospital,Third Military Medical University,Xinqiao St

【出 处】

：

第二十四届长城国际心脏病学会议、亚太心脏大会暨国际心血管病预防与康复会议(The 24th Great Wall Int

【发表日期】

：

2013年2期

下载到本地 , 更方便阅读

下载此文 赞助VIP

声明 : 本文档内容版权归属内容提供方 , 如果您对本文有版权争议 , 可与客服联系进行内容授权或下架

论文部分内容阅读

Objectives Syncope is caused by a wide variety of disorders.Recurrent syncope as a complication of malignancy is uncommon and may be difficult to diagnose and to treat.Methods In such cases, surgery, chemotherapy or radiotherapy can be performed, because syncope may be an early sign of pharynx cancer.

其他文献

EXPRESSION AND ANTI-APOPTOSIS ROLE OF MICRORNA-21 IN RAT MYOCARDIUM DURING EARLY ISCHAEMIA-REPERFUSI

Objectives To observe the expression and anti-apoptosis role of microRNA-21 (miR-21) in rat myocardium during early ischaemia-reperfusion injury (I/R).Methods Sprague-Dawley rats were randomly divided

会议

THE EFFECTS OF RUANMAILING ON THE REORGANISATION OF COFILIN, TWINFILIN-1 AND PROFILIN-1 INDUCED BY P

Objectives To observe the effect of Ruanmailing component of traditional Chinese medicine on the reorganisation of cofilin,twinfilin-1, profilin-1 induced by platelet-derived growth factor (PDGF) in v

会议

GEMININ INTERFERENCE FACILITATE VASCULAR SMOOTH MUSCLE CELLS PROLIFERATION VIA UPREGULATION OF CDT1

Objectives Silencing Geminin gene selectively by using RNA interference and investigate the mechanism of the Geminin gene silence regulating VSMCs proliferation.Methods VSMCs proliferation were determ

会议

TIROFIBAN IMPROVES RENAL OUTCOME IN A RAT MODEL OF ISCHAEMIA/REPERFUSION INJURY BY MODULATING NO SYN

Objectives Renal ischaemia/reperfusion (I/R) injury is a common clinical disease.We have known that NO-signal transduction has an important effect on renal I/R injury.NO is produced by NO synthases.En

会议

STIMULATION OF D3 RECEPTOR ATTENUATES RENAL ISCHAEMIA/REPERFUSION INJURY VIA INCREASED ITS LINKAGE W

Objectives Renal ischaemia reperfusion injury (IRI) causes renal tubular necrosis, apoptosis and inflammation leading to acute kidney dysfunction.Recent studies suggest knockout of Gα12 protects from

会议

EFFECT OF DOPAMINE D3 RECEPTORS ON MIGRATION OF VASCULAR SMOOTH MUSCLE CELL INDUCED BY INSULIN-LIKE

Objectives To investigate the stimulative effect of D3 dopamine receptors on the insulin-like growth factors-1 (IGF-1)-induced vascular smooth muscle cells (VSMCs) migration and the mechanism underlyi

会议

INVOLVEMENT OF VASCULAR PEROXIDASE 1 IN ANGIOTENSIN II-INDUCED H9C2 CELLS HYPERTROPHY

Objectives Oxidative stress induces hypertrophic gene expression and collagen deposition that mediates cardiac hypertrophy and myocardial fibrosis.Vascular peroxidase 1 (VPO1) is a new heme-containing

会议

1S-LIPOXYGENASE-1 ACTIVITY: INVOLVE IN THE PATHWAY OF ENDOTHELIAL FUNCTION IMPROVEMENT BY ATORVASTAT

Objectives 15-Lipoxygenase-1(15-LO) oxidises polyunsaturated fatty acids to a rich spectrum of biologically active metabolites,and its deregulation has been reported involved in the development of ath

会议

EFFECTS OF VALSARTAN ON EXPRESSION OF GP130 IN CARDIAC HYPERTROPHY RAT INDUCED BY ANG Ⅱ

Objectives To observe effects of valsartan on expression of gp130 in cardiac hypertrophy rat induced by Ang Ⅱ.Methods Neonatal rat cardiomyocyte induced by Ang Ⅱ into cardiac hypertrophy, and cultivat

会议

HYPERHOMOCYSTEINEMIA IMPAIR ANGIOGENESIS VIA HGF INHIBITION

Objectives Abundant epidemiological and clinical studies have revealed the close relationship between hyperhomocysteinemia (HHcy) and the increased frequency of CVD.During the last decade, we and othe

会议