流行病学和组织病理学研究指出幽门螺杆菌（ Hp） 感染和胃癌发生正相关， 1994年世界卫生组织将 Hp定为Ⅰ类致癌因子。细胞凋亡是受基因编码的细胞主动“自杀”的生理性死亡过程，肿瘤的发生与细胞凋亡异常有关。 Hp感染主要在胃癌发生的起始阶段起作用 [1]，尽管人群 Hp感染率 Epidemiological and histopathological studies indicate that Helicobacter pylori (Hp) infection is positively correlated with gastric cancer. In 1994, the World Health Organization set Hp as a class I oncogene. Apoptosis is a process of physiological death which is actively “suicide” by gene-encoded cells. The occurrence of tumors is related to abnormal apoptosis. Hp infection mainly in the initial stage of gastric cancer play a role [1], although the rate of Hp infection