肾脏的钾排泄象氯化钠一样,肾脏改变钾排泄的能力在很大的范围变动。例如,慢性钾耗竭的存在,尿钾浓度可减少至5mEq/L,而摄入钾增加时,尿钾浓度可达100mEq/L或更多。虽然钾绝大部分可自由地在肾小球滤过,但血浆钾量小与非饱和的血浆蛋白有关(并将因此对过滤无效)。肾小球滤过率每天达150公升和4mEq/L的血浆钾浓度,每天钾的滤过负荷将是600mEq左右。滤过的钾 Kidney potassium excretion, like sodium chloride, changes the kidney’s ability to excrete potassium over a wide range. For example, the presence of chronic potassium depletion reduces urinary potassium concentrations to 5 mEq / L and increases urinary potassium up to 100 mEq / L or more with increased potassium intake. Although potassium is largely free glomerular filtration, plasma potassium is less associated with non-saturated plasma proteins (and will therefore be ineffective against filtration). Glomerular filtration rate of 150 liters per day and 4mEq / L of plasma potassium concentration, potassium per day filter load will be about 600mEq. Filtered potassium