败血症性休克常继发于肺炎、亚急性细菌性心内膜炎、脏器破裂性腹膜炎以及腹腔脓肿等败血症过程。革兰氏阴性和阳性细菌被吞噬细胞吞噬后,在细胞内溶酶体酶作用下,细菌细胞的胞浆蛋白和膜蛋白发生降解,细菌的外毒素或内毒素(细胞壁的脂蛋白多糖)释放入血。已证实,革兰氏阴性细菌壁的脂蛋白多 Septic shock often secondary to pneumonia, subacute bacterial endocarditis, organ rupture peritonitis and abdominal sepsis and other sepsis process. Gram-negative and positive bacteria are phagocytosed by phagocytes, the cytoplasmic and membrane proteins of bacterial cells are degraded by intracellular lysosomal enzymes, and release of bacterial exotoxins or endotoxins (lipoprotein polysaccharide from cell walls) Into the blood. It has been demonstrated that there are many lipoproteins in Gram-negative bacterial walls