论文部分内容阅读
在肺部疾病、肺梗塞、吸气、心脏停搏过程中及在高原条件下,心脏可处于低氧状态。有慢性肺部疾病的病人,在运动和睡眠时处于严重的低氧状态。夜间低氧的机理是复杂的,包括肺泡通气的减少和通气、血流比值失调。在这些病人中,低氧导致了继发性肺高压和肺心病。有睡眠呼吸暂停综合征的病人,无论是周围性或中枢性,也存在有夜间低氧的情况。动脉低血氧与心肌缺血对心脏作用不同点的研究很少。其不同点可能为:首先,动脉低氧血症是全身性的而心肌缺血是局限的;其次,缺血心肌的细胞代谢不同于充分灌注而低血氧的心肌。
In lung disease, pulmonary infarction, inspiratory, cardiac arrest and in the plateau conditions, the heart can be in hypoxia. Patients with chronic lung disease are at severe hypoxia during exercise and sleep. The mechanism of nocturnal hypoxia is complex, including reduction of alveolar ventilation and ventilation, imbalance of blood flow ratio. In these patients, hypoxia led to secondary pulmonary hypertension and cor pulmonale. Patients with sleep apnea syndrome, either peripheral or central, also have night nocturnal hypoxia. Artery hypoxia and myocardial ischemia on the role of the heart of different studies. The differences may be: First, arterial hypoxemia is systemic and myocardial ischemia is limited; secondly, the cellular metabolism of ischemic myocardium differs from that of fully perfusing hypoxemic myocardium.