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血栓性微血管病(TMA)分血栓性血小板减少性紫癜及溶血尿毒症综合征(HUS),HUS又分感染后HUS和非典型HUS,现认为HUS发病主要与补体旁路途径的过度激活相关。
Thrombotic thrombocytopenia (TMA) thrombolytic thrombocytopenic purpura and hemolytic uremic syndrome (HUS), HUS sub-infected HUS and atypical HUS, HUS pathogenesis is believed to be mainly associated with excessive activation of complement bypass pathway.