神经管缺陷为人类的一种先天性疾病 ,发病率高。这类患儿除有双下肢功能障碍外 ,常伴有腹胀、便秘等。除已知的括约肌功能障碍外 ,是否还存在胃肠道动力减低以及相应的神经机制改变 ,目前还未见报道。本实验目的是利用神经管缺陷鸡胚模型 ,观察病变小肠ＮＡＤＰＨ 黄递酶组织化 Neural tube defects as a human congenital diseases, the incidence of high. In addition to these children with lower extremity dysfunction, often accompanied by abdominal distension, constipation and so on. In addition to the known sphincter dysfunction, there is still a decrease in gastrointestinal motility and the corresponding changes in neural mechanisms have not been reported. The purpose of this experiment is to use neural tube defects chicken embryo model to observe the lesion small intestine NADPH diaphorase organization