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本文报告应用1,6-二磷酸果糖治疗阵发性室上性心动过速4例,3例即时效果显著,1例无效。其作用机理为通过心肌ATP之释放,再转化为腺苷酸而终止室上速。本品安全有效。其真正机理与使用剂量等尚须进一步观察与探讨。
This report reports the application of fructose 1,6 - diphosphate treatment of paroxysmal supraventricular tachycardia in 4 cases, 3 cases of immediate effect was significant, 1 case of ineffective. Its mechanism of action is through the release of myocardial ATP, and then converted to adenosine and termination of supraventricular tachycardia. This product is safe and effective. Its true mechanism and dosage need to be further observed and discussed.