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本实验主要研究在缺氧过程中表皮生长因子受体(EGFR)激酶活化的机制,并验证在新生小猪大脑皮层神经细胞膜缺氧导致的EGFR激酶活性的增加是否由神经型一氧化氮合酶(nNOS)产生的一氧化氮(NO)
This experiment mainly studied the mechanism of EGFR kinase activation during hypoxia and verified whether the increase of EGFR kinase activity induced by hypoxia in neuronal cell membrane of neonate pigs was induced by neuronal nitric oxide synthase (nNOS) -produced nitric oxide (NO)