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三例新生儿和三例胎儿尾状核的血脑屏障超微结构无明显差异.它们的毛细血管内皮有胞饮泡;内皮有无隔膜的孔,内皮连接较短而直.内皮可有连续、完整的基膜,厚约40～50nm,但也有的内皮无连续基膜,只见到絮状、电子密度低的基膜样物质,胶质膜环绕,或未完全环绕内皮外周.胶质膜上可见到胞饮作用.新生儿尾状核中血脑屏障超微结构特点,使新生儿尾状核中血脑屏障通透性较大.在某种病理条件下,如缺氧、酸中毒破坏血脑屏障,促进血液中游离胆红素越过血脑屏障,沉积于脑基底核,引起新生儿核黄疸.
There was no significant difference in the ultrastructure of the blood-brain barrier in three newborns and three fetal caudate nuclei, with capillaries with capillaries in their endothelial endothelium, with or without septa in the endothelium with shorter and straight endothelial connections. , A complete basement membrane, about 40 ~ 50nm thick, but some have no continuous basement membrane, only to see flocculent, electron density of the basement membrane-like substances, glial membrane around, or not completely surround the outer periphery of the glial The membrane can be seen on the role of pinocytosis neonatal caudate nucleus in the ultrastructure of the blood-brain barrier, neonatal caudate nucleus in the blood-brain barrier permeability greater under certain pathological conditions, such as hypoxia, acid Poisoning damage the blood-brain barrier, promote free bilirubin in the blood across the blood-brain barrier, deposition in the basal ganglia, causing neonatal kernicterus.