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为探讨正常孕妇和妊高征患者的血液动力学参数，采用无创妊高征血液动力学监测系统（ＭＰ－ＰＩＨ），序贯监测孕早、中、晚期正常孕妇３０例，非孕妇６０例，妊高征（轻、中、重度）患者４６例。非孕妇和正常孕妇属正常排，微循环、流量、流速、流态正常，两者无明显差异。妊高征正常排：ＣＩ（２．８５±０．３９）Ｌ．ｍｉｎ－１／ｍ２，ＡＬＫ（０．０４±０．０１）１／ｓ，ＡＬＴ（１９．５６±２．４６）ｓ，ＴＭ（２８．２３±３．５５）ｓ，与正常孕妇无显著差异。高排低阻：ＣＩ（４．８４±０．４１）Ｌ·ｍｉｎ－１／ｍ２，ＡＬＫ（０．０６±０．０１）１／ｓ，ＡＬＴ（１１．４２±０．９５）ｓ、ＴＭ（１６．４８±１．３５）ｓ。心排量＞４Ｌ·ｍｉｎ－１／ｍ２，表明心肌代偿性加强收缩，血流速加快经微循环直捷通路，缺乏与组织细胞间的营养、代谢交换。低排高阻：ＣＩ（２．０９±０．２８）Ｌ·ｍｉｎ－１／ｍ２，ＡＬＫ（０．０３±０）１／ｓ，ＡＬＴ（２６．７８±４．０２）ｓ，ＴＭ（３８．６５±５．７６）ｓ。心排量＜２．５Ｌ·ｍｉｎ－１／ｍ２，血流量少，流速慢，微循环淤血，组织细胞缺血、缺氧。
In order to investigate the hemodynamic parameters of normal pregnancy and PIH patients, non-invasive pregnancy-induced hypertension hemodynamic monitoring system (MP-PIH) was used to sequentially monitor 30 cases of normal pregnant women in early, middle and late pregnancy, 60 non-pregnant women, PIH (mild, moderate and severe) patients in 46 cases. Non-pregnant women and normal pregnant women is a normal row, microcirculation, flow, velocity, normal flow, no significant difference between the two. PIH normal row: CI (2.85 ± 0.39) L. ALT (19.56 ± 2.46) s, TM (28.23 ± 3.55) s, no significant difference compared with normal pregnant women . High row and low resistance: CI (4.84 ± 0.41) L · min-1 / m2, ALK (0.06 ± 0.01) 1 / s, ALT (11.42 ± 0.95) s, TM (16.48 ± 1.35) s. Cardiac output> 4L · min-1 / m2, indicating that compensatory myocardial contraction to enhance blood flow faster through the microcirculation of the direct access, lack of nutrition and tissue cells between the exchange of metabolism. Low-row high resistance: CI (2.09 ± 0.28) L · min -1 / m2, ALK 0.03 0.03, ALT 26.78 ± 4.02, TM 38 .65 ± 5.76) s. Cardiac discharge <2.5L · min-1 / m2, less blood flow, slow flow, microcirculation congestion, tissue ischemia, hypoxia.