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为了分析急性低氧时心泵代偿-失代偿转变过程的力学特征,在22条麻醉开胸狗上,于吸入气氧分压递降条件下,作了心泵舒缩功能测定。结果表明,虽然严重低氧时心泵外功功率尚可保持在增大状态,但其压力作功成分减小,容积作功成分增大。动脉导纳的增加是容积作功增大的主要原因。这时,主动脉内有效血液推动力进一步增大,而泵缩力量却开始减弱。肌力效应在泵失代偿发生前,对舒缩功能的增强起着重要作用。在泵代偿-失代偿转变阶段,斯塔林效应的作用显著。“舒-缩谐调”的变化,早在泵失代偿前就已出现,并渐明显,值得注意。
In order to analyze the mechanics characteristics of cardiac pump compensation-decompensation process in acute hypoxia, heart-pump systolic and diastolic function was measured in 22 anesthetized thoracotics dogs under the condition of decreasing inhaled oxygen and partial pressure. The results showed that, although severe hypoxia in cardiac pump external power can still maintain an increase in the state, but the pressure work composition decreases, the volume of work composition increases. The increase of arterial admittance is the main reason for the increase of volume. At this moment, the effective blood driving force in the aorta further increases, but the pumping power begins to weaken. Muscle effect in the loss of pump before the occurrence of contraction and contraction function plays an important role in enhancing. In the pump compensation - decompensation stage of change, the role of Starling effect is significant. “Shu - shrink tone” changes, as long as the pump before the loss has appeared, and became apparent, it is noteworthy.