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本文报道中药活血化瘀煎剂在临床与实验室证实可阻止溶血的基础上，为探讨其作用机制，对G6PD缺乏与G6PD正常新生儿进行治疗前后G6PD／6PGD比值对比观察，结果活血化瘀煎剂服后均可使G6PD缺乏者G6PD／6PDD比值增高，甚至达正常，而G6PD正常者服后比值不变，提示活血化瘀煎剂作用机制可能基于强大之抗氧化作用及抗骨髓休克、活跃生血，促进G6PD之生成，甚至纠正其缺乏，而不仅是替代作用。为开发此方剂作为广泛防治G6PD缺乏病儿，提供了科学依据。
This article reports on the basis of clinical and laboratory confirmed that hemorrhage decoction can prevent hemolysis, in order to explore its mechanism of action, G6PD deficiency and G6PD normal neonates before and after treatment G6PD / 6PGD ratio comparison, the results of blood circulation of blood stasis G6PD / 6PDD ratio could be increased or even up to normal in G6PD deficiency patients after oral administration, while the ratio of G6PD in normal service after taking the same dosage suggests that the mechanism of action of activating blood circulation and removing blood stasis decoction may be based on powerful anti-oxidation and anti-bone marrow shock. Blood, promote the formation of G6PD, and even correct its lack, not just a substitute effect. For the development of this prescription as a comprehensive prevention and treatment of G6PD deficiency children, provides a scientific basis.