支气管哮喘的发病机理,目前认为是免疫球蛋白E抗体与肥大细胞、嗜硷性白细胞或可能嗜酸性白细胞的细胞膜上的特异受体结合,而使这些细胞致敏,此后,特异抗原和细胞膜上的免疫球蛋白E抗体结合,使这些细胞释放出化学介质如组织胺及慢反应物 The pathogenesis of bronchial asthma is currently considered to be the binding of immunoglobulin E antibodies to specific receptors on the membranes of mast cells, erythematocytic leukocytes, or possibly eosinophils, which sensitizes these cells, and thereafter, on specific antigens and cell membranes. The binding of immunoglobulin E antibodies allows these cells to release chemical mediators such as histamine and slow reagents