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实验在３１只兔身上进行。利用自制的刺激电极和微量灌注管对核团进行刺激和微量灌注。观察到一侧孤束核（ＮＴＳ）和脑干网状结构尾端腹外侧区（ｃＶＬＭ）内微量注入ｒ－氨基丁酸（ＧＡＢＡ）后，电刺激ＮＴＳ引起肾交感神经放电反应（ＮＴＳ－ＳＮＤ）的抑制效应明显减弱，可持续１０～１５分钟。说明在兔ＮＴＳ和ｃＶＬＭ中含有对肾交感神经放电和血压调控的ＧＡＢＡ能系统。而ＮＴＳ对交感神经放电抑制和使血压下降至少部分是通过ｃＶＬＭ中的ＧＡＢＡ受体发挥作用的。
Experiments were performed on 31 rabbits. Nuclei were stimulated and microinjected using self-made stimulation electrodes and micropipettes. NTS-SND (NTS-SND) was induced by electrical stimulation of NTS after microinjection of r-aminobutyric acid (GABA) into the nucleus tractus solitarius (NTS) and cVLM ) Inhibitory effect was significantly weakened, sustainable 10 to 15 minutes. Description GABAergic system containing renal sympathetic nerve discharge and blood pressure regulation in rabbit NTS and cVLM. Whereas NTS suppresses sympathetic discharge and decreases blood pressure at least in part through GABA receptors in cVLM.