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血管紧张素的转换是肺内皮细胞重要功能之一,成人呼吸窘迫综合征(ARDS)时肺内皮细胞损伤,可引起血液和肺灌洗液中血管紧张素转换酶(angiotensin-converting enzyme,ACE)变化。本文检测36例ARDS血ACE,其中败血性19例,非败血性17例。同时检测补体系统活性、血凝状态、呼吸功能。发现在第1个24小时内,62％病人ACE降低,38％正常。败血性和非败血性ARDS两者无差异。败血性与非败血性各7例作动态观察血ACE8天。11例ACE降低,第72～96小时降至最低,第
Angiotensin-converting is one of the important functions of pulmonary endothelial cells. Injury of lung endothelial cells in adult respiratory distress syndrome (ARDS) can cause angiotensin-converting enzyme (ACE) in blood and lung lavage fluid, Variety. In this paper, 36 cases of ARDS blood ACE, including sepsis in 19 cases, 17 cases of non-sepsis. At the same time, detection of complement system activity, coagulation status, respiratory function. In the first 24 hours, ACE decreased in 62% of patients and was 38% normal. There was no difference in septic and non-septic ARDS. Septicemia and non-septicemia in each of 7 cases of dynamic observation of blood ACE8 days. 11 cases of ACE decreased, 72 to 96 hours to a minimum, the first