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内皮细胞收缩和细胞连接间隙开放是炎症介质引起血管壁通透性增高的主要机制。我们利用以APOLLODN 3500为主机的图像工作站建立了内皮细胞形态参数分析的软件,对血小板激活因子(PAF)引起的内皮细胞面积、形状因子、细胞间距及细胞间空隙面积占细胞单层面积百分比等形态参数变化进行定量分析,并研究了
Endothelium contraction and cell gap junction opening is the main mechanism by which inflammatory mediators cause increased vascular wall permeability. We use the APOLLODN 3500 as the host image workstation to establish the endothelial cell morphological parameters of the software, the platelet-activating factor (PAF) -induced endothelial cell area, shape factor, cell spacing and intercellular space area accounts for the percentage of cell monolayer area The morphological parameters were quantitatively analyzed and studied