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目的 探讨白内障形成的病因。方法 通过扫描电镜技术 ,对 1 0例老年性白内障和 5例正常人的晶状体皮质纤维表面结构进行了观察和分析。结果 正常晶体状体纤维排列整齐 ,均匀一致 ,纤维间以各种突起相互连接。随着层次的加深 ,纤维变扁平 ,突起明显减少。老年性白内障组晶体皮质纤维表面结构与正常人相比有很大差异 ,明显的变化是纤维排列紊乱 ,纤维溶解、融合。突起的肿胀、变形、脱落、崩溃以及变性球样小体的出现。结论 随年龄增长晶状体可溶性蛋白减少 ,不溶性蛋白增加 ,蛋白由高移低级蛋白变性向液化转变。说明白内障的形成与蛋白变性有关。
Objective To investigate the etiology of cataract formation. Methods The surface structure of cortical fibers in 10 cases of senile cataract and 5 normal subjects were observed and analyzed by scanning electron microscopy. Results Normal crystalline fibers were arranged neatly and uniformly, and the fibers were connected with each other by various protrusions. With the deepening of the hierarchy, the fibers become flattened and the protrusions are significantly reduced. The surface structure of cortical fiber in the senile cataract group is quite different from the normal one. The obvious change is the disordered fiber arrangement, fiber dissolution and fusion. Protrusion swelling, deformation, loss, collapse and the appearance of degeneration ball-like bodies. Conclusion With age, the number of soluble protein in lens decreases, the amount of insoluble protein increases, and the protein changes from high to low level of protein to liquefaction. Explain the formation of cataract and protein degeneration related.